Preliminary experiments reveal conolidine may possibly inhibit certain ion channels, lessening neuronal excitability and restricting suffering signals. This system is particularly applicable in neuropathic suffering, wherever abnormal signaling leads to persistent pain. Moreover, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to pain notion. Comput... https://michelangelox095vps2.wikinewspaper.com/user