Preliminary scientific studies suggest conolidine may perhaps inhibit certain ion channels, minimizing neuronal excitability and restricting discomfort signals. This mechanism is especially suitable in neuropathic suffering, in which abnormal signaling triggers persistent pain. In addition, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to discomfort no... https://brucef061tka7.wikifrontier.com/user